View article

A. has been shown to develop acquired resistance to a bacterial and a fungal pathogen following treatment with the activator compound 2,6-dichloroisonicotinic acid (INA). The necrotizing pathogen, turnip crinkle carmovirus (TCV) induced systemic acquired resistance (SAR) in A. thaliana against bacterial (Pseudomonas syringae) and viral (TCV) pathogens. The TCV-induced resistance results in both a decrease in visible disease symptoms and a reduction in the growth of the challenge pathogen. Levels of salicylic acid (SA), a likely endogenous signal for SAR, increased more than fivefold in response to TCV infection. In addition, exogenously applied SA induced resistance to the fungal pathogen Peronospora parasitica. Three genes induced to high levels by SA (PR-1, PR-2 and PR-5) were also induced by TCV inoculation. In addition, it is shown that INA treatment induces resistance to TCV, extending the …