Authors
Gkikas Magiorkinis, Konstantinos Angelis, Ioannis Mamais, Aris Katzourakis, Angelos Hatzakis, Jan Albert, Glenn Lawyer, Osamah Hamouda, Daniel Struck, Jurgen Vercauteren, Annemarie Wensing, Ivailo Alexiev, Birgitta Åsjö, Claudia Balotta, Perpétua Gomes, Ricardo J Camacho, Suzie Coughlan, Algirdas Griskevicius, Zehava Grossman, Anders Horban, Leondios G Kostrikis, Snjezana J Lepej, Kirsi Liitsola, Marek Linka, Claus Nielsen, Dan Otelea, Roger Paredes, Mario Poljak, Elizabeth Puchhammer-Stöckl, Jean Claude Schmit, Anders Sönnerborg, Danica Staneková, Maja Stanojevic, Dora C Stylianou, Charles AB Boucher, Georgios Nikolopoulos, Tetyana Vasylyeva, Samuel R Friedman, David van de Vijver, Gioacchino Angarano, Marie-Laure Chaix, Andrea De Luca, Klaus Korn, Clive Loveday, Vincent Soriano, Sabine Yerly, Mauricio Zazzi, Anne-Mieke Vandamme, Dimitrios Paraskevis
Publication date
2016/12/1
Journal
Infection, genetics and evolution
Volume
46
Pages
169-179
Publisher
Elsevier
Description
Human immunodeficiency virus type 1 (HIV-1) was discovered in the early 1980s when the virus had already established a pandemic. For at least three decades the epidemic in the Western World has been dominated by subtype B infections, as part of a sub-epidemic that traveled from Africa through Haiti to United States. However, the pattern of the subsequent spread still remains poorly understood. Here we analyze a large dataset of globally representative HIV-1 subtype B strains to map their spread around the world over the last 50 years and describe significant spread patterns. We show that subtype B travelled from North America to Western Europe in different occasions, while Central/Eastern Europe remained isolated for the most part of the early epidemic. Looking with more detail in European countries we see that the United Kingdom, France and Switzerland exchanged viral isolates with non-European …
Scholar articles
G Magiorkinis, K Angelis, I Mamais, A Katzourakis… - Infection, genetics and evolution, 2016