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Available somewhere: 48
Interpreting cancer genomes using systematic host network perturbations by tumour virus proteins
O Rozenblatt-Rosen, RC Deo, M Padi, G Adelmant, MA Calderwood, ...
Nature 487 (7408), 491-495, 2012
Mandates: US National Institutes of Health
Genome-wide analysis reveals conserved and divergent features of Notch1/RBPJ binding in human and murine T-lymphoblastic leukemia cells
H Wang, J Zou, B Zhao, E Johannsen, T Ashworth, H Wong, WS Pear, ...
Proceedings of the National Academy of Sciences 108 (36), 14908-14913, 2011
Mandates: US National Institutes of Health, Howard Hughes Medical Institute
Epstein-Barr virus exploits intrinsic B-lymphocyte transcription programs to achieve immortal cell growth
B Zhao, J Zou, H Wang, E Johannsen, C Peng, J Quackenbush, JC Mar, ...
Proceedings of the National Academy of Sciences 108 (36), 14902-14907, 2011
Mandates: US National Institutes of Health
Epstein-Barr virus oncoprotein super-enhancers control B cell growth
H Zhou, SCS Schmidt, S Jiang, B Willox, K Bernhardt, J Liang, ...
Cell host & microbe 17 (2), 205-216, 2015
Mandates: US National Institutes of Health, Wellcome Trust
Epstein-Barr virus nuclear antigens 3C and 3A maintain lymphoblastoid cell growth by repressing p16INK4A and p14ARF expression
S Maruo, B Zhao, E Johannsen, E Kieff, J Zou, K Takada
Proceedings of the National Academy of Sciences 108 (5), 1919-1924, 2011
Mandates: US National Institutes of Health
Epigenetics of human papillomaviruses
E Johannsen, PF Lambert
Virology 445 (1-2), 205-212, 2013
Mandates: US National Institutes of Health
Epstein-Barr virus LF2: an antagonist to type I interferon
L Wu, E Fossum, CH Joo, KS Inn, YC Shin, E Johannsen, ...
Journal of virology 83 (2), 1140-1146, 2009
Mandates: US National Institutes of Health
Epstein–Barr Virus Nuclear Antigen 3C binds to BATF/IRF4 or SPI1/IRF4 composite sites and recruits Sin3A to repress CDKN2A
S Jiang, B Willox, H Zhou, AM Holthaus, A Wang, TT Shi, S Maruo, ...
Proceedings of the National Academy of Sciences 111 (1), 421-426, 2014
Mandates: US National Institutes of Health
Differentiation-dependent KLF4 expression promotes lytic Epstein-Barr virus infection in epithelial cells
DM Nawandar, A Wang, K Makielski, D Lee, S Ma, E Barlow, J Reusch, ...
PLoS pathogens 11 (10), e1005195, 2015
Mandates: US Department of Energy, US National Institutes of Health
A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection
JA Bristol, R Djavadian, ER Albright, CB Coleman, M Ohashi, M Hayes, ...
PLoS pathogens 14 (7), e1007179, 2018
Mandates: US National Institutes of Health, UK Medical Research Council
Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infection
AM Price, J Dai, Q Bazot, L Patel, PA Nikitin, R Djavadian, PS Winter, ...
Elife 6, e22509, 2017
Mandates: US National Institutes of Health, Wellcome Trust
EBV nuclear antigen EBNALP dismisses transcription repressors NCoR and RBPJ from enhancers and EBNA2 increases NCoR-deficient RBPJ DNA binding
D Portal, B Zhao, MA Calderwood, T Sommermann, E Johannsen, E Kieff
Proceedings of the National Academy of Sciences 108 (19), 7808-7813, 2011
Mandates: US National Institutes of Health
Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus’s natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early …
RJ Kraus, X Yu, BLA Cordes, S Sathiamoorthi, T Iempridee, ...
PLoS pathogens 13 (6), e1006404, 2017
Mandates: US National Institutes of Health
Epstein–Barr virus nuclear antigen 3C regulated genes in lymphoblastoid cell lines
B Zhao, JC Mar, S Maruo, S Lee, BE Gewurz, E Johannsen, K Holton, ...
Proceedings of the National Academy of Sciences 108 (1), 337-342, 2011
Mandates: US National Institutes of Health
The EBNA3 family of Epstein-Barr virus nuclear proteins associates with the USP46/USP12 deubiquitination complexes to regulate lymphoblastoid cell line growth
M Ohashi, AM Holthaus, MA Calderwood, CY Lai, B Krastins, D Sarracino, ...
PLoS pathogens 11 (4), e1004822, 2015
Mandates: US National Institutes of Health
An Epstein-Barr Virus-Encoded Protein Complex Requires an Origin of Lytic Replication In Cis to Mediate Late Gene Transcription
R Djavadian, YF Chiu, E Johannsen
PLoS pathogens 12 (6), e1005718, 2016
Mandates: US National Institutes of Health
Epstein–Barr virus nuclear antigen 3A partially coincides with EBNA3C genome-wide and is tethered to DNA through BATF complexes
SCS Schmidt, S Jiang, H Zhou, B Willox, AM Holthaus, PV Kharchenko, ...
Proceedings of the National Academy of Sciences 112 (2), 554-559, 2015
Mandates: US National Institutes of Health
Genome-wide analysis of Epstein-Barr virus Rta DNA binding
AMF Heilmann, MA Calderwood, D Portal, Y Lu, E Johannsen
Journal of virology 86 (9), 5151-5164, 2012
Mandates: US National Institutes of Health, Howard Hughes Medical Institute, German …
The Epstein-Barr virus LF2 protein inhibits viral replication
MA Calderwood, AM Holthaus, E Johannsen
Journal of virology 82 (17), 8509-8519, 2008
Mandates: US National Institutes of Health, Howard Hughes Medical Institute
Differentiation-dependent LMP1 expression is required for efficient lytic Epstein-Barr virus reactivation in epithelial cells
DM Nawandar, M Ohashi, R Djavadian, E Barlow, K Makielski, A Ali, ...
Journal of virology 91 (8), 10.1128/jvi. 02438-16, 2017
Mandates: US National Institutes of Health
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